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Stroke and Vascular Neurology

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Open Access

Current management of spontaneous intracerebral haemorrhage

Cyrus K Dastur, Wengui Yu
DOI: 10.1136/svn-2016-000047 Published 24 February 2017
Cyrus K Dastur
Department of Neurology, University of California Irvine, Irvine, California, USA
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Wengui Yu
Department of Neurology, University of California Irvine, Irvine, California, USA
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    Figure 1

    Typical locations of hypertensive ICH are putamen (A), thalamus (B), subcortical white matter (C), pons (D) and cerebellum (E). Thalamic and subcortical haemorrhages often extend into ventricles (B and C). CAA, drug abuse or vascular anomaly often causes lobar haemorrhage (F). ICH, intracerebral haemorrhage; CAA, cerebral amyloid angiopathy.

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    Figure 2

    The ED algorithm for early diagnosis and emergent intervention. ICH, intracerebral haemorrhage.

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    Figure 3

    The guidelines for reversing warfarin and NOAC coagulopathies in patients with symptomatic ICH. ICH, intracerebral haemorrhage.

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Vol 2 Issue 1 Table of Contents
Stroke and Vascular Neurology: 2 (1)
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Current management of spontaneous intracerebral haemorrhage
Cyrus K Dastur, Wengui Yu
Stroke and Vascular Neurology Mar 2017, 2 (1) 21-29; DOI: 10.1136/svn-2016-000047

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Current management of spontaneous intracerebral haemorrhage
Cyrus K Dastur, Wengui Yu
Stroke and Vascular Neurology Mar 2017, 2 (1) 21-29; DOI: 10.1136/svn-2016-000047
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Current management of spontaneous intracerebral haemorrhage
Cyrus K Dastur, Wengui Yu
Stroke and Vascular Neurology Mar 2017, 2 (1) 21-29; DOI: 10.1136/svn-2016-000047
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  • Article
    • Abstract
    • Introduction
    • Classification
    • Early diagnosis
    • Emergent intervention
    • Reversal of coagulopathies
    • Surgical intervention
    • Management of perihaematoma oedema
    • Seizure prophylaxis and treatment
    • Glucose management
    • Fever control
    • Deep vein thrombosis prophylaxis and treatment
    • Prognosis
    • Conclusions
    • Footnotes
    • References
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